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Author Topic: Oleylethanolamide, Satiety, Love and the Endocannabinoid system  (Read 1123 times)

Offline Chip (OP)

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... So i was thinking about better ways of losing weight and after reviewing a lots of studies on brain fats, i came across OleylethanolamideWiki but as of yet, have not purchased and not have i tried it.

Here is some info that i found:

Endocannabinoid systemWiki

(look it up, it's bountiful)

The satiety signal oleoylethanolamide stimulates oxytocin neurosecretion from rat hypothalamic neurons.

at https://www.ncbi.nlm.nih.gov/pubmed/23959001

Quote
Abstract

The anandamide monounsaturated analogue oleoylethanolamide (OEA) acts as satiety signal released from enterocytes upon the ingestion of dietary fats to prolong the interval to the next meal. This effect, which requires intact vagal fibers and intestinal PPAR-alpha receptors, is coupled to the increase of c-fos and oxytocin mRNA expression in neurons of the paraventricular nucleus (PVN) and is prevented by the intracerebroventricular administration of a selective oxytocin antagonist, thus suggesting a necessary role of oxytocinergic neurotransmission in the pro-satiety effect of OEA. By brain microdialysis and immunohistochemistry, in this study we demonstrate that OEA treatment can stimulate oxytocin neurosecretion from the PVN and enhance oxytocin expression at both axonal and somatodendritic levels of hypothalamic neurons. Such effects, which are maximum 2h after OEA administration, support the hypothesis that the satiety-inducing action of OEA is mediated by the activation of oxytocin hypothalamic neurons.
« Last Edit: May 27, 2019, 11:23:11 PM by smfadmin »
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I do not condone or support any illegal activities. All information is for theoretical discussion and wonder.
All activities discussed are considered fictional and hypothetical. Information of all discussion has been derived from online research and in the spirit of personal Freedom.

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